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Although our cerebral GSH results do not support the redox hypothesis of autism in the brain, previous results of decreased GSH in the plasma of autistic children may help explain some other inflammatory symptoms that autistic children experience (e.g

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Figure 3 In the hypoxic microenvironment, HIF dimers can bind to the co-activator p300/CBP (CREB-binding protein) to form a complex, which binds to the promoter region of the target gene containing the hypoxia response element (HRE) to promote its transcriptional expression (16)