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(2014) reported that TNF -1031 polymorphism is a possible risk factor for PD, and Wang et al
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Human SOD1 ‐linked familial ALS patients with an A4V mutation typically exhibit early onset, rapidly progressing ALS, [161] whereas in mice the expression of a SOD1 A4V transgene elicits a slow progressing form of ALS which develops comparatively late in life
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