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wirkung von l carnitin L-Carnitin: und Einnahme phospholipid
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It has been proposed that in the setting of MSH2 Lynch syndrome, the adenomas may evolve through the classic pathway or from flat dMMR crypts (perhaps through an activating mutation in β-catenin rather than an inactivating mutation in APC ) and the subsequent accumulation of somatic mutations leads to the emergence of a polypoid lesion that can be removed through surveillance, as occurs also in the setting of MSH6 and PMS2 Lynch syndrome (Figure 3a, 3b) 35
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Acknowledgements This work was supported by grants from National Basic Research Program of China (2015CB910600)
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