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Hyperhomocysteinemia: An Independent Risk Factor for Thrombosis Recent studies underline the major role of moderate hyperhomocysteinemia on the odds of developing both arterial and venous thromboembolisms (Boers, 1997a
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(60) utilized next generation sequencing (NGS) and computational analysis to demonstrate that miR-23b-3p, acting as a tumor suppressor, can inhibit tumor Gln metabolism by targeting GLS1, thereby enhancing the sensitivity of liver tumor cells to sorafenib
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