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The resulting metabolic dysregulation includes impaired thermogenesis, altered glucose and lipid metabolism, and a tendency toward insulin resistance—factors that also contribute to MetS and increased CVD risk [40,41]

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↑ "The isoniazid-NAD adduct is a slow, tight-binding inhibitor of InhA, the Mycobacterium tuberculosis enoyl reductase: Adduct affinity and drug resistance"