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The Many Facets of PPAR-γ Agonism in Obesity and Associated Comorbidities: Benefits, Risks, Challenges, and Future Directions | Current Obesity Reports | Springer Nature Link
Another possible reason is that tumour hypoxemia resulting from anti–VEGF-A/VEGFR therapy lead to the over-expression of matrix components that bind and sequester VEGF-A, rendering anti-VEGF drugs ineffective [86]
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The Arabidopsis thaliana salt tolerance gene SOS1 encodes a putative Na + /H + antiporter
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