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Similarly, in melanoma, the overexpression of a protein called UT-B was shown to increase NO levels, leading to mitochondrial depolarization, a spike in ROS, and reduced cell viability

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Although the role of exercise in regulating PNPLA7 and further PINK1/Parkin recruitment remains unexplored, studies have shown that metabolically active tissues such as skeletal muscle, adipose tissue, and the liver are highly expressed for PNPLA7, which dynamically responds to energy availability and metabolic stress (Ji et al., 2020)